Wilson Cui, MD, PhD

Assistant Clinical Professor
Adult Cardiothoracic Anesthesia Fellowship Director
Publications: 
Cui WW, Ramsay JG. Pharmacologic approaches to weaning from cardiopulmonary bypass and extracorporeal membrane oxygenation. Best Pract Res Clin Anaesthesiol. 2015 Jun; 29(2):257-70.
Salehi A, Cui WW. Aorta-right atrial tunnel in an elderly patient. Anesth Analg. 2013 Dec; 117(6):1282-5.
Horstick EJ, Linsley JW, Dowling JJ, Hauser MA, McDonald KK, Ashley-Koch A, Saint-Amant L, Satish A, Cui WW, Zhou W, Sprague SM, Stamm DS, Powell CM, Speer MC, Franzini-Armstrong C, Hirata H, Kuwada JY. Stac3 is a component of the excitation-contraction coupling machinery and mutated in Native American myopathy. Nat Commun. 2013; 4:1952.
Hirata H, Wen H, Kawakami Y, Naganawa Y, Ogino K, Yamada K, Saint-Amant L, Low SE, Cui WW, Zhou W, Sprague SM, Asakawa K, Muto A, Kawakami K, Kuwada JY. Connexin 39.9 protein is necessary for coordinated activation of slow-twitch muscle and normal behavior in zebrafish. J Biol Chem. 2012 Jan 06; 287(2):1080-9.
Low SE, Amburgey K, Horstick E, Linsley J, Sprague SM, Cui WW, Zhou W, Hirata H, Saint-Amant L, Hume RI, Kuwada JY. TRPM7 is required within zebrafish sensory neurons for the activation of touch-evoked escape behaviors. J Neurosci. 2011 Aug 10; 31(32):11633-44.
Low SE, Ryan J, Sprague SM, Hirata H, Cui WW, Zhou W, Hume RI, Kuwada JY, Saint-Amant L. touché Is required for touch-evoked generator potentials within vertebrate sensory neurons. J Neurosci. 2010 Jul 14; 30(28):9359-67.
Low SE, Zhou W, Choong I, Saint-Amant L, Sprague SM, Hirata H, Cui WW, Hume RI, Kuwada JY. Na(v)1.6a is required for normal activation of motor circuits normally excited by tactile stimulation. Dev Neurobiol. 2010 Jun; 70(7):508-22.
Saint-Amant L, Sprague SM, Hirata H, Li Q, Cui WW, Zhou W, Poudou O, Hume RI, Kuwada JY. The zebrafish ennui behavioral mutation disrupts acetylcholine receptor localization and motor axon stability. Dev Neurobiol. 2008 Jan; 68(1):45-61.
Hirata H, Watanabe T, Hatakeyama J, Sprague SM, Saint-Amant L, Nagashima A, Cui WW, Zhou W, Kuwada JY. Zebrafish relatively relaxed mutants have a ryanodine receptor defect, show slow swimming and provide a model of multi-minicore disease. Development. 2007 Aug; 134(15):2771-81.
Zhou W, Saint-Amant L, Hirata H, Cui WW, Sprague SM, Kuwada JY. Non-sense mutations in the dihydropyridine receptor beta1 gene, CACNB1, paralyze zebrafish relaxed mutants. Cell Calcium. 2006 Mar; 39(3):227-36.
Cui WW, Low SE, Hirata H, Saint-Amant L, Geisler R, Hume RI, Kuwada JY. The zebrafish shocked gene encodes a glycine transporter and is essential for the function of early neural circuits in the CNS. J Neurosci. 2005 Jul 13; 25(28):6610-20.
Hirata H, Saint-Amant L, Downes GB, Cui WW, Zhou W, Granato M, Kuwada JY. Zebrafish bandoneon mutants display behavioral defects due to a mutation in the glycine receptor beta-subunit. Proc Natl Acad Sci U S A. 2005 Jun 07; 102(23):8345-50.
Hirata H, Saint-Amant L, Waterbury J, Cui W, Zhou W, Li Q, Goldman D, Granato M, Kuwada JY. accordion, a zebrafish behavioral mutant, has a muscle relaxation defect due to a mutation in the ATPase Ca2+ pump SERCA1. Development. 2004 Nov; 131(21):5457-68.
Cui WW, Saint-Amant L, Kuwada JY. shocked Gene is required for the function of a premotor network in the zebrafish CNS. J Neurophysiol. 2004 Nov; 92(5):2898-908.