Role Faculty Location UCSF at Mission Bay UCSF at Mount Zion UCSF at Parnassus Category Clinical Specialties Adult Cardiac Liver Transplant Education 2018 - Diversity, Equity, and Inclusion Champion Training, University of CaliforniaFellowship, 2013 - Cardiothoracic Anesthesia, University of California, Los AngelesResidency, 2012 - Anesthesiology, University of California, San FranciscoInternship, 2009 - , Evanston Hospital, NorthwesternM.D., Ph.D., 2008 - , University of Michigan Medical SchoolB.S., 1999 - Chemistry/Biochemistry, University of California, San Diego Publications Pharmacologic approaches to weaning from cardiopulmonary bypass and extracorporeal membrane oxygenation. Cui WW, Ramsay JG Aorta-right atrial tunnel in an elderly patient. Salehi A, Cui WW Stac3 is a component of the excitation-contraction coupling machinery and mutated in Native American myopathy. Horstick EJ, Linsley JW, Dowling JJ, Hauser MA, McDonald KK, Ashley-Koch A, Saint-Amant L, Satish A, Cui WW, Zhou W, Sprague SM, Stamm DS, Powell CM, Speer MC, Franzini-Armstrong C, Hirata H, Kuwada JY Connexin 39.9 protein is necessary for coordinated activation of slow-twitch muscle and normal behavior in zebrafish. Hirata H, Wen H, Kawakami Y, Naganawa Y, Ogino K, Yamada K, Saint-Amant L, Low SE, Cui WW, Zhou W, Sprague SM, Asakawa K, Muto A, Kawakami K, Kuwada JY TRPM7 is required within zebrafish sensory neurons for the activation of touch-evoked escape behaviors. Low SE, Amburgey K, Horstick E, Linsley J, Sprague SM, Cui WW, Zhou W, Hirata H, Saint-Amant L, Hume RI, Kuwada JY touché Is required for touch-evoked generator potentials within vertebrate sensory neurons. Low SE, Ryan J, Sprague SM, Hirata H, Cui WW, Zhou W, Hume RI, Kuwada JY, Saint-Amant L Na(v)1.6a is required for normal activation of motor circuits normally excited by tactile stimulation. Low SE, Zhou W, Choong I, Saint-Amant L, Sprague SM, Hirata H, Cui WW, Hume RI, Kuwada JY The zebrafish ennui behavioral mutation disrupts acetylcholine receptor localization and motor axon stability. Saint-Amant L, Sprague SM, Hirata H, Li Q, Cui WW, Zhou W, Poudou O, Hume RI, Kuwada JY Zebrafish relatively relaxed mutants have a ryanodine receptor defect, show slow swimming and provide a model of multi-minicore disease. Hirata H, Watanabe T, Hatakeyama J, Sprague SM, Saint-Amant L, Nagashima A, Cui WW, Zhou W, Kuwada JY Non-sense mutations in the dihydropyridine receptor beta1 gene, CACNB1, paralyze zebrafish relaxed mutants. Zhou W, Saint-Amant L, Hirata H, Cui WW, Sprague SM, Kuwada JY The zebrafish shocked gene encodes a glycine transporter and is essential for the function of early neural circuits in the CNS. Cui WW, Low SE, Hirata H, Saint-Amant L, Geisler R, Hume RI, Kuwada JY Zebrafish bandoneon mutants display behavioral defects due to a mutation in the glycine receptor beta-subunit. Hirata H, Saint-Amant L, Downes GB, Cui WW, Zhou W, Granato M, Kuwada JY accordion, a zebrafish behavioral mutant, has a muscle relaxation defect due to a mutation in the ATPase Ca2+ pump SERCA1. Hirata H, Saint-Amant L, Waterbury J, Cui W, Zhou W, Li Q, Goldman D, Granato M, Kuwada JY shocked Gene is required for the function of a premotor network in the zebrafish CNS. Cui WW, Saint-Amant L, Kuwada JY
